Cerebral Aneurysms: Microvascular and Endovascular by Robert R. Smith M.D., Yuri N. Zubkov M.D., Yahgoub Tarassoli

By Robert R. Smith M.D., Yuri N. Zubkov M.D., Yahgoub Tarassoli M.D. (auth.)

Surgical fix of cerebral aneurysms is a center element of neurosurgical perform. whereas open microvascular strategy has ruled Western surgical perform, surgeons within the former USSR have built endovascular innovations that experience received attractiveness between surgeons right here. this article demonstrates either surgical and endovascular techniques, written and illustrated via surgeons with significant event in either, in a comparative context. The paintings is broadly illustrated with full-color surgical illustrations, line drawings, and radiographs.

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Additional resources for Cerebral Aneurysms: Microvascular and Endovascular Management

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SAH causes a distinct clinical pattern that correlates with the amount of blood present in the subarachnoid pathways. Those with cerebro-subarachnoid hemorrhage (Cerebro-SAH) can be separated into two distinct classes based on the volume of the hematoma present in the brain. Those in whom the hematoma dissects into the ventricular system also have predictable clinical courses and outcomes dependent upon tamponade. Associated Disorders of the Hemorrhagic Period Associated Disorders of the Hemorrhagic Period The causes of the deterioration in SAH relate to vasospasm, hydrocephalus, intracranial hypertension, and coagulation disorders.

57. Kudrjashov BA. Modern condition knowledge about the antihemostatic system of blood. Probl Hematol Blood Transfusion. 1962;12:3-13. 58. Crompton MR. Cerebral infarction following the rupture of cerebral berry aneurysms. Brain. 1969;87:263-279. 59. Antunes JL, Correll JW. Cerebral emboli from intracranial aneurysms. Surg Neurol. 1976;6:7-10. 60. Auld A W, Shafey S. Transient ischemic attacks not produced by extracranial vascular disease. A plea for complete and early angiographic investigation.

Hematomas occupying the fourth ventricle and the aqueduct of Sylvius are often due to retrograde propulsion of blood through the subarachnoid pathways. In these cases, with retrograde dissection, severe acute hydrocephalus due to tamponade of the cisterna magna and major basal cisterns invariably occurs. Clinical signs depend on the location of the hematoma, and its size. Commonly, hematomas develop near the central sulcus and thus, motor and sensory disorders, and speech problems arise if the hematoma is in the dominant hemisphere.

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